Murine ORAI2 Splice Variants Form Functional Ca2+ Release-activated Ca2+ (CRAC) Channels
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference29 articles.
1. A severe defect in CRAC Ca2+ channel activation and altered K+ channel gating in T cells from immunodeficient patients
2. A mutation in Orai1 causes immune deficiency by abrogating CRAC channel function
3. CRACM1 Is a Plasma Membrane Protein Essential for Store-Operated Ca 2+ Entry
4. Genome-wide RNAi screen of Ca2+ influx identifies genes that regulate Ca2+ release-activated Ca2+ channel activity
5. Orai1 is an essential pore subunit of the CRAC channel
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