Oxidative Stress-mediated, Post-translational Loss of MafA Protein as a Contributing Mechanism to Loss of Insulin Gene Expression in Glucotoxic Beta Cells-Reference-Cited by-同舟云学术

Oxidative Stress-mediated, Post-translational Loss of MafA Protein as a Contributing Mechanism to Loss of Insulin Gene Expression in Glucotoxic Beta Cells

Published:2005-03 Issue:12 Volume:280 Page:11107-11113
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Harmon Jamie S.,Stein Roland,Robertson R. Paul

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference32 articles.

1. Prolonged exposure of human pancreatic islets to high glucose concentrations in vitro impairs the beta-cell function.

2. Preservation of insulin mRNA levels and insulin secretion in HIT cells by avoidance of chronic exposure to high glucose concentrations.

3. Beneficial effects of antioxidants in diabetes: possible protection of pancreatic beta-cells against glucose toxicity.

4. Prevention of glucose toxicity in HIT-T15 cells and Zucker diabetic fatty rats by antioxidants

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