Differential Activation of Protein Kinase B and p70S6K by Glucose and Insulin-like Growth Factor 1 in Pancreatic β-Cells (INS-1)-Reference-Cited by-同舟云学术

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Differential Activation of Protein Kinase B and p70S6K by Glucose and Insulin-like Growth Factor 1 in Pancreatic β-Cells (INS-1)

Published:2001-06 Issue:24 Volume:276 Page:21110-21120
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Dickson Lorna M.,Lingohr Melissa K.,McCuaig Jill,Hügl Sigrun R.,Snow Lynn,Kahn Barbara B.,Myers Martin G.,Rhodes Christopher J.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference49 articles.

1. Persistent reduction of pancreatic Beta-cell mass after a limited period of protein-energy malnutrition in the young rat

2. IGF-I and GH post-receptor signaling mechanisms for pancreatic beta-cell replication

3. Disruption of IRS-2 causes type 2 diabetes in mice

4. Stimulation of pancreatic β-cell proliferation by growth hormone is glucose-dependent: signal transduction via Janus kinase 2 (JAK2)/signal transducer and activator of transcription 5 (STAT5) with no crosstalk to insulin receptor substrate-mediated mitogenic signalling

5. Insulin-like Growth Factor I (IGF-I)-stimulated Pancreatic β-Cell Growth Is Glucose-dependent

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2. Prolonged Exposure to Insulin Inactivates Akt and Erk1/2 and Increases Pancreatic Islet and INS1E β-Cell Apoptosis;Journal of the Endocrine Society;2018-11-01

3. Characterization of somatostatin receptors and associated signaling pathways in pancreas of R6/2 transgenic mice;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2018-02

4. Nuclear Export of FoxO1 Is Associated with ERK Signaling in β-Cells Lacking Insulin Receptors;Journal of Biological Chemistry;2016-10

5. Glucose-Induced Oxidative Stress Reduces Proliferation in Embryonic Stem Cells via FOXO3A/β-Catenin-Dependent Transcription of p21cip1;Stem Cell Reports;2016-07

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