Mutant Presenilins Disturb Neuronal Calcium Homeostasis in the Brain of Transgenic Mice, Decreasing the Threshold for Excitotoxicity and Facilitating Long-term Potentiation-Reference-Cited by-同舟云学术

Mutant Presenilins Disturb Neuronal Calcium Homeostasis in the Brain of Transgenic Mice, Decreasing the Threshold for Excitotoxicity and Facilitating Long-term Potentiation

Published:2001-04 Issue:15 Volume:276 Page:11539-11544
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Schneider Ilka,Reversé Delphine,Dewachter Ilse,Ris Laurence,Caluwaerts Nathalie,Kuipéri Cuno,Gilis Martine,Geerts Hugo,Kretzschmar Hans,Godaux Emile,Moechars Dieder,Van Leuven Fred,Herms Jochen

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference49 articles.

1. Aging Increased Amyloid Peptide and Caused Amyloid Plaques in Brain of Old APP/V717I Transgenic Mice by a Different Mechanism than Mutant Presenilin1

2. Prominent Cerebral Amyloid Angiopathy in Transgenic Mice Overexpressing the London Mutant of Human APP in Neurons

3. Amyloid, the presenilins and Alzheimer's disease

4. The cell biology of β-amyloid precursor protein and presenilin in Alzheimer's disease

5. Cloning of a gene bearing missense mutations in early-onset familial Alzheimer's disease

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