The Kindler Syndrome Protein Is Regulated by Transforming Growth Factor-β and Involved in Integrin-mediated Adhesion
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference67 articles.
1. Identification of mutations in a new gene encoding a FERM family protein with a pleckstrin homology domain in Kindler syndrome
2. URP1: a member of a novel family of PH and FERM domain-containing membrane-associated proteins is significantly over-expressed in lung and colon carcinomas
3. Loss of Kindlin-1, a Human Homolog of the Caenorhabditis elegans Actin–Extracellular-Matrix Linker Protein UNC-112, Causes Kindler Syndrome
4. Structural Determinants of Integrin Recognition by Talin
5. Leaving the neighborhood: molecular mechanisms involved during epithelial-mesenchymal transition
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