A β-Arrestin-dependent Scaffold Is Associated with Prolonged MAPK Activation in Pseudopodia during Protease-activated Receptor-2-induced Chemotaxis
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference36 articles.
1. A Cell's Sense of Direction
2. Phosphorylation of WAVE Downstream of Mitogen-activated Protein Kinase Signaling
3. Regulation of Cell Motility by Mitogen-activated Protein Kinase
4. ERK and RhoA Differentially Regulate Pseudopodia Growth and Retraction during Chemotaxis
5. β-Arrestin2 Is Critically Involved in CXCR4-mediated Chemotaxis, and This Is Mediated by Its Enhancement of p38 MAPK Activation
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