Fibroblast-specific Expression of a Kinase-deficient Type II Transforming Growth Factor β (TGFβ) Receptor Leads to Paradoxical Activation of TGFβ Signaling Pathways with Fibrosis in Transgenic Mice-Reference-Cited by-同舟云学术

Fibroblast-specific Expression of a Kinase-deficient Type II Transforming Growth Factor β (TGFβ) Receptor Leads to Paradoxical Activation of TGFβ Signaling Pathways with Fibrosis in Transgenic Mice

Published:2003-07 Issue:27 Volume:278 Page:25109-25119
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Denton Christopher P.,Zheng Bing,Evans Lowri A.,Shi-wen Xu,Ong Voon H.,Fisher Ivan,Lazaridis Konstantinos,Abraham David J.,Black Carol M.,de Crombrugghe Benoit

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference61 articles.

1. Biology of TGF-β in knockout and transgenic mouse models

2. TGF-β Receptor Type II Deficiency Results in Defects of Yolk Sac Hematopoiesis and Vasculogenesis

3. Making sense of latent TGFβ activation

4. Thrombospondin-1 Is a Major Activator of TGF-β1 In Vivo

5. TGF-beta induces fibronectin synthesis through a c-Jun N-terminal kinase-dependent, Smad4-independent pathway

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