Overexpression of Protein-tyrosine Phosphatase-1B in Adipocytes Inhibits Insulin-stimulated Phosphoinositide 3-Kinase Activity without Altering Glucose Transport or Akt/Protein Kinase B Activation-Reference-Cited by-同舟云学术

Overexpression of Protein-tyrosine Phosphatase-1B in Adipocytes Inhibits Insulin-stimulated Phosphoinositide 3-Kinase Activity without Altering Glucose Transport or Akt/Protein Kinase B Activation

Published:2000-06 Issue:24 Volume:275 Page:18318-18326
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Venable Carol L.,Frevert Ernst U.,Kim Young-Bum,Fischer Britta M.,Kamatkar Shubhangi,Neel Benjamin G.,Kahn Barbara B.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference52 articles.

1. Type 2 Diabetes: When Insulin Secretion Fails to Compensate for Insulin Resistance

2. The insulin signalling system and the IRS proteins

3. From receptor to transporter: insulin signalling to glucose transport

4. A Novel 160-kDa Phosphotyrosine Protein in Insulin-treated Embryonic Kidney Cells Is a New Member of the Insulin Receptor Substrate Family

5. The 60-kDa Phosphotyrosine Protein in Insulin-treated Adipocytes Is a New Member of the Insulin Receptor Substrate Family

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