Insulin-like Growth Factor-binding Protein-3 Modulates Expression of Bax and Bcl-2 and Potentiates p53-independent Radiation-induced Apoptosis in Human Breast Cancer Cells-Reference-Cited by-同舟云学术

Insulin-like Growth Factor-binding Protein-3 Modulates Expression of Bax and Bcl-2 and Potentiates p53-independent Radiation-induced Apoptosis in Human Breast Cancer Cells

Published:2000-12 Issue:50 Volume:275 Page:39174-39181
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Butt Alison J.,Firth Sue M.,King Malcolm A.,Baxter Robert C.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference51 articles.

1. Growth hormone-dependent insulin-like growth factor (IGF) binding protein both inhibits and potentiates IGF-I-stimulated DNA synthesis in human skin fibroblasts

2. Potentiation of insulin-like growth factor (IGF) action by IGF-binding protein-3: studies of underlying mechanism.

3. Insulin-like growth factor-binding protein enhancement of insulin-like growth factor-i (IGF-I)-mediated DNA synthesis and IGF-I binding in a human breast carcinoma cell line

4. The insulin-like growth factor binding proteins (IGFBPs) in human breast cancer

5. Insulin-like growth factor (IGF)-independent action of IGF-binding protein-3 in Hs578T human breast cancer cells. Cell surface binding and growth inhibition

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