A Rac1/Phosphatidylinositol 3-Kinase/Akt3 Anti-apoptotic Pathway, Triggered by AlsinLF, the Product of the ALS2 Gene, Antagonizes Cu/Zn-superoxide Dismutase (SOD1) Mutant-induced Motoneuronal Cell Death-Reference-Cited by-同舟云学术

A Rac1/Phosphatidylinositol 3-Kinase/Akt3 Anti-apoptotic Pathway, Triggered by AlsinLF, the Product of the ALS2 Gene, Antagonizes Cu/Zn-superoxide Dismutase (SOD1) Mutant-induced Motoneuronal Cell Death

Published:2005-02 Issue:6 Volume:280 Page:4532-4543
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Kanekura Kohsuke,Hashimoto Yuichi,Kita Yoshiko,Sasabe Jumpei,Aiso Sadakazu,Nishimoto Ikuo,Matsuoka Masaaki

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference57 articles.

1. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

2. Motor Neuron Degeneration in Mice that Express a Human Cu,Zn Superoxide Dismutase Mutation

3. Restricted Expression of G86R Cu/Zn Superoxide Dismutase in Astrocytes Results in Astrocytosis But Does Not Cause Motoneuron Degeneration

4. Mutant SOD1 causes motor neuron disease independent of copper chaperone–mediated copper loading

5. A gene encoding a putative GTPase regulator is mutated in familial amyotrophic lateral sclerosis 2

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