Selective Intracellular Release of Copper and Zinc Ions from Bis(thiosemicarbazonato) Complexes Reduces Levels of Alzheimer Disease Amyloid-β Peptide-Reference-Cited by-同舟云学术

Selective Intracellular Release of Copper and Zinc Ions from Bis(thiosemicarbazonato) Complexes Reduces Levels of Alzheimer Disease Amyloid-β Peptide

Published:2008-02 Issue:8 Volume:283 Page:4568-4577
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Donnelly Paul S.,Caragounis Aphrodite,Du Tai,Laughton Katrina M.,Volitakis Irene,Cherny Robert A.,Sharples Robyn A.,Hill Andrew F.,Li Qiao-Xin,Masters Colin L.,Barnham Kevin J.,White Anthony R.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference37 articles.

1. Cognitive decline correlates with low plasma concentrations of copper in patients with mild to moderate Alzheimer's disease

2. In vivo reduction of amyloid- by a mutant copper transporter

3. Dietary Cu stabilizes brain superoxide dismutase 1 activity and reduces amyloid A production in APP23 transgenic mice

4. Trace amounts of copper in water induce -amyloid plaques and learning deficits in a rabbit model of Alzheimer's disease

5. Copper and Alzheimer's disease

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