β-Arrestin-mediated Angiotensin II Signaling Controls the Activation of ARF6 Protein and Endocytosis in Migration of Vascular Smooth Muscle Cells-Reference-Cited by-同舟云学术

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β-Arrestin-mediated Angiotensin II Signaling Controls the Activation of ARF6 Protein and Endocytosis in Migration of Vascular Smooth Muscle Cells

Published:2016-02 Issue:8 Volume:291 Page:3967-3981
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Charles Ricardo,Namkung Yoon,Cotton Mathieu,Laporte Stéphane A.,Claing Audrey

Funder

Canadian Institutes of Health Research

Heart and Stroke Foundation of Canada

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference68 articles.

1. Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone;Rajagopalan;J. Clin. Invest,1996

2. Role of superoxide in angiotensin II-induced but not catecholamine-induced hypertension;Laursen;Circulation,1997

3. p22phox is a critical component of the superoxide-generating NADH/NADPH oxidase system and regulates angiotensin II-induced hypertrophy in vascular smooth muscle cells;Ushio-Fukai;J. Biol. Chem,1996

4. β-Arrestin 2-dependent angiotensin II type 1A receptor-mediated pathway of chemotaxis;Hunton;Mol. Pharmacol,2005

5. p38 mitogen-activated protein kinase contributes to angiotensin II-stimulated migration of rat aortic smooth muscle cells;Lee;J. Pharmacol. Sci,2007

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