Endogenous Ligand for GPR120, Docosahexaenoic Acid, Exerts Benign Metabolic Effects on the Skeletal Muscles via AMP-activated Protein Kinase Pathway-Reference-Cited by-同舟云学术

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Endogenous Ligand for GPR120, Docosahexaenoic Acid, Exerts Benign Metabolic Effects on the Skeletal Muscles via AMP-activated Protein Kinase Pathway

Published:2015-08 Issue:33 Volume:290 Page:20438-20447
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Kim Nami,Lee Jung Ok,Lee Hye Jeong,Kim Hyung Ip,Kim Joong Kwan,Lee Yong Woo,Lee Soo Kyung,Kim Su Jin,Park Sun Hwa,Kim Hyeon Soo

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference43 articles.

1. The AMP-activated protein kinase–fuel gauge of the mammalian cell?;Hardie;Eur. J. Biochem,1997

2. Deficiency of LK1 in skeletal muscle prevents AMPK activation and glucose uptake during contraction;Sakamoto;EMBO J,2005

3. Skeletal muscle-selective knockout of LKB1 increases insulin sensitivity, improves glucose homeostasis, and decreases TRB3;Koh;Mol. Cell. Biol,2006

4. Ca2+/calmodulin-dependent protein kinase kinase-beta acts upstream of AMP-activated protein kinase in mammalian cells;Woods;Cell Metab,2005

5. Calmodulin-dependent protein kinase kinase-beta is an alternative upstream kinase for AMP-activated protein kinase;Hawley;Cell Metab,2005

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