Impaired Ca2+ Store Functions in Skeletal and Cardiac Muscle Cells from Sarcalumenin-deficient Mice-Reference-Cited by-同舟云学术

Impaired Ca2+ Store Functions in Skeletal and Cardiac Muscle Cells from Sarcalumenin-deficient Mice

Published:2005-02 Issue:5 Volume:280 Page:3500-3506
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Yoshida Morikatsu,Minamisawa Susumu,Shimura Miei,Komazaki Shinji,Kume Hideaki,Zhang Miao,Matsumura Kiyoyuki,Nishi Miyuki,Saito Minori,Saeki Yasutake,Ishikawa Yoshihiro,Yanagisawa Teruyuki,Takeshima Hiroshi

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference21 articles.

1. Calcium Release from Sarcoplasmic Reticulum

2. Ca2+ signalling and muscle disease

3. Calsequestrin Is an Inhibitor of Skeletal Muscle Ryanodine Receptor Calcium Release Channels

4. A Retrograde Signal from Calsequestrin for the Regulation of Store-operated Ca2+ Entry in Skeletal Muscle

5. Purification, calcium binding properties, and ultrastructural localization of the 53,000- and 160,000 (sarcalumenin)-dalton glycoproteins of the sarcoplasmic reticulum.

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