Inactivation of Tumor Suppressor p53 by Mot-2, a hsp70 Family Member-Reference-Cited by-同舟云学术

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Inactivation of Tumor Suppressor p53 by Mot-2, a hsp70 Family Member

Published:1998-11 Issue:45 Volume:273 Page:29586-29591
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Wadhwa Renu,Takano Syuichi,Robert Martin,Yoshida Akiko,Nomura Hitoshi,Reddel Roger R.,Mitsui Youji,Kaul Sunil C.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference37 articles.

1. p53, the Cellular Gatekeeper for Growth and Division

2. Suppression of Human Colorectal Carcinoma Cell Growth by Wild-Type p53

3. p53 alteration is a common event in the spontaneous immortalization of primary BALB/c murine embryo fibroblasts.

4. Absence of p53 allows direct immortalization of hematopoietic cells by the myc and raf oncogenes

5. Reinitiation of DNA Synthesis and Cell Division in Senescent Human Fibroblasts by Microinjection of Anti-p53 Antibodies

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1. Restoration of p53 functions by suppression of mortalin–p53 sequestration: an emerging target in cancer therapy;Future Medicinal Chemistry;2023-11

2. Unveiling the Intercompartmental Signaling Axis: Mitochondrial to ER Stress Response (MERSR) and its Impact on Proteostasis;2023-09-08

3. Identification of an Imidazopyridine-based Compound as an Oral Selective Estrogen Receptor Degrader for Breast Cancer Therapy;Cancer Research Communications;2023-07-27

4. Identification of a new member of Mortaparib class of inhibitors that target mortalin and PARP1;Frontiers in Cell and Developmental Biology;2022-09-12

5. The mitochondrial unfolded protein response (UPRmt): shielding against toxicity to mitochondria in cancer;Journal of Hematology & Oncology;2022-07-21

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