MK2 Targets AU-rich Elements and Regulates Biosynthesis of Tumor Necrosis Factor and Interleukin-6 Independently at Different Post-transcriptional Levels-Reference-Cited by-同舟云学术

MK2 Targets AU-rich Elements and Regulates Biosynthesis of Tumor Necrosis Factor and Interleukin-6 Independently at Different Post-transcriptional Levels

Published:2002-02 Issue:5 Volume:277 Page:3065-3068
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Neininger Armin,Kontoyiannis Dimitris,Kotlyarov Alexey,Winzen Reinhard,Eckert Rolf,Volk Hans-Dieter,Holtmann Helmut,Kollias George,Gaestel Matthias

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference28 articles.

1. The p38 signal transduction pathway Activation and function

2. p38 Mitogen-Activated Protein Kinase Inhibitors— Mechanisms and Therapeutic Potentials

3. Deficiency of the Stress Kinase P38α Results in Embryonic Lethality

4. Defective IL-12 production in mitogen-activated protein (MAP) kinase kinase 3(Mkk3)-deficient mice

5. Requirement of mitogen-activated protein kinase kinase 3 (MKK3) for tumor necrosis factor-induced cytokine expression

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