R-Ras Glucosylation and Transient RhoA Activation Determine the Cytopathic Effect Produced by Toxin B Variants from Toxin A-negative Strains of Clostridium difficile
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference37 articles.
1. Effects of Clostridium difficile toxins given intragastrically to animals
2. Toxins A and B from Clostridium difficile differ with respect to enzymatic potencies, cellular substrate specificities, and surface binding to cultured cells.
3. Clostridium difficile Toxins A and B Are Cation-dependent UDP-glucose Hydrolases with Differing Catalytic Activities
4. Glucosylation of Rho proteins by Clostridium difficile toxin B
5. The Enterotoxin from Clostridium difficile (ToxA) Monoglucosylates the Rho Proteins
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