Neurofibromatosis Type I Tumor Suppressor Neurofibromin Regulates Neuronal Differentiation via Its GTPase-activating Protein Function toward Ras-Reference-Cited by-同舟云学术

Neurofibromatosis Type I Tumor Suppressor Neurofibromin Regulates Neuronal Differentiation via Its GTPase-activating Protein Function toward Ras

Published:2003-07 Issue:29 Volume:278 Page:26958-26969
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Yunoue Shunji,Tokuo Hiroshi,Fukunaga Kohji,Feng Liping,Ozawa Tatsuya,Nishi Toru,Kikuchi Akira,Hattori Seisuke,Kuratsu Junichi,Saya Hideyuki,Araki Norie

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference42 articles.

1. Linkage studies with chromosome 17 DNA markers in 45 neurofibromatosis 1 families

2. A major segment of the neurofibromatosis type 1 gene: cDNA sequence, genomic structure, and point mutations

3. Deletions and a translocation interrupt a cloned gene at the neurofibromatosis type 1 locus

4. The NF1 locus encodes a protein functionally related to mammalian GAP and yeast IRA proteins

5. The catalytic domain of the neurofibromatosis type 1 gene product stimulates ras GTPase and complements ira mutants of S. cerevisiae

Cited by 76 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Mechanistic insights from animal models of neurofibromatosis type 1 cognitive impairment;Disease Models & Mechanisms;2022-08-01

2. Alterations in Molecular Profiles Affecting Glioblastoma Resistance to Radiochemotherapy: Where Does the Good Go?;Cancers;2022-05-13

3. An Assessment of the Therapeutic Landscape for the Treatment of Heart Disease in the RASopathies;Cardiovascular Drugs and Therapy;2022-02-14

4. Neurofibromin and suppression of tumorigenesis: beyond the GAP;Oncogene;2022-01-23

5. Rationale for haploinsufficiency correction therapy in neurofibromatosis type 1;Journal of Translational Genetics and Genomics;2022