Chloride Channel (Clc)-5 Is Necessary for Exocytic Trafficking of Na+/H+ Exchanger 3 (NHE3)-Reference-Cited by-同舟云学术

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Chloride Channel (Clc)-5 Is Necessary for Exocytic Trafficking of Na+/H+ Exchanger 3 (NHE3)

Published:2011-07 Issue:26 Volume:286 Page:22833-22845
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Lin Zhihong,Jin Shi,Duan Xiaohong,Wang Tong,Martini Sabrina,Hulamm Phuson,Cha Boyoung,Hubbard Ann,Donowitz Mark,Guggino Sandra E.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference49 articles.

1. Dent's disease, a renal Fanconi syndrome with nephrocalcinosis and kidney stones, is associated with a microdeletion involving DXS255 and maps to Xp11.22

2. ClC-5 Cl--channel disruption impairs endocytosis in a mouse model for Dent's disease

3. Mice lacking renal chloride channel, CLC-5, are a model for Dent's disease, a nephrolithiasis disorder associated with defective receptor-mediated endocytosis

4. High citrate diet delays progression of renal insufficiency in the ClC-5 knockout mouse model of Dent's disease

5. Mechanisms of Disease: what can mouse models tell us about the molecular processes underlying Dent disease?

Cited by 21 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Impaired Endosome Maturation Mediates Tubular Proteinuria in Dent Disease Cell Culture and Mouse Models;Journal of the American Society of Nephrology;2023-02-09

2. Megalin, a multi-ligand endocytic receptor, and its participation in renal function and diseases: A review;Life Sciences;2022-11

3. Megalin: a Novel Determinant of Renin-Angiotensin System Activity in the Kidney?;Current Hypertension Reports;2020-03-14

4. Genetic Analyses in Dent Disease and Characterization of CLCN5 Mutations in Kidney Biopsies;International Journal of Molecular Sciences;2020-01-14

5. Making a Dent in Dent Disease;Function;2020

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