Carbohydrate Metabolism Is Perturbed in Peroxisome-deficient Hepatocytes Due to Mitochondrial Dysfunction, AMP-activated Protein Kinase (AMPK) Activation, and Peroxisome Proliferator-activated Receptor γ Coactivator 1α (PGC-1α) Suppression-Reference-Cited by-同舟云学术

Carbohydrate Metabolism Is Perturbed in Peroxisome-deficient Hepatocytes Due to Mitochondrial Dysfunction, AMP-activated Protein Kinase (AMPK) Activation, and Peroxisome Proliferator-activated Receptor γ Coactivator 1α (PGC-1α) Suppression

Published:2011-12 Issue:49 Volume:286 Page:42162-42179
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Peeters Annelies,Fraisl Peter,van den Berg Sjoerd,Ver Loren van Themaat Emiel,Van Kampen Antoine,Rider Mark H.,Takemori Hiroshi,van Dijk Ko Willems,Van Veldhoven Paul P.,Carmeliet Peter,Baes Myriam

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference66 articles.

1. Biochemistry of Mammalian Peroxisomes Revisited

2. Peroxisomes and oxidative stress

3. Peroxisome biogenesis disorders

4. Histopathologic Approach to Metabolic Liver Disease: Part 1

5. Peroxisomal and Mitochondrial Defects in the Cerebro-Hepato-Renal Syndrome

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