Guanine Nucleotide Exchange Factor-like Factor (Rlf) Induces Gene Expression and Potentiates α1-Adrenergic Receptor-induced Transcriptional Responses in Neonatal Rat Ventricular Myocytes-Reference-Cited by-同舟云学术

Guanine Nucleotide Exchange Factor-like Factor (Rlf) Induces Gene Expression and Potentiates α1-Adrenergic Receptor-induced Transcriptional Responses in Neonatal Rat Ventricular Myocytes

Published:2002-05 Issue:18 Volume:277 Page:15286-15292
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Post Ginell R.,Swiderski Carol,Waldrop Bruce A.,Salty Lina,Glembotski Christopher C.,Wolthuis Rob M.F.,Mochizuki Naoki

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference61 articles.

1. Regulation of cardiac gene expression during myocardial growth and hypertrophy: molecular studies of an adaptive physiologic response

2. HRas-dependent pathways can activate morphological and genetic markers of cardiac muscle cell hypertrophy.

3. Ventricular Expression of a MLC-2v-ras Fusion Gene Induces Cardiac Hypertrophy and Selective Diastolic Dysfunction in Transgenic Mice

4. M1Muscarinic Receptors Heterologously Expressed in Cardiac Myocytes Mediate Ras-dependent Changes in Gene Expression

5. Pathways and roadblocks in muscarinic receptor-mediated growth regulation

Cited by 10 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Literature-Based Discovery to Elucidate the Biological Links between Resistant Hypertension and COVID-19;Biology;2023-09-21

2. Cardiac Alpha1-Adrenergic Receptors: Novel Aspects of Expression, Signaling Mechanisms, Physiologic Function, and Clinical Importance;Pharmacological Reviews;2013-12-24

3. Small G Proteins in the Cardiovascular System: Physiological and Pathological Aspects;Physiological Reviews;2013-10

4. The Ral Exchange Factor Rgl2 Promotes Cardiomyocyte Survival and Inhibits Cardiac Fibrosis;PLoS ONE;2013-09-17

5. RalGDS family members couple Ras to Ral signalling and that's not all;Cellular Signalling;2010-12