ERp29 Regulates ΔF508 and Wild-type Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Trafficking to the Plasma Membrane in Cystic Fibrosis (CF) and Non-CF Epithelial Cells-Reference-Cited by-同舟云学术

ERp29 Regulates ΔF508 and Wild-type Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Trafficking to the Plasma Membrane in Cystic Fibrosis (CF) and Non-CF Epithelial Cells

Published:2011-06 Issue:24 Volume:286 Page:21239-21253
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Suaud Laurence,Miller Katelyn,Alvey Lora,Yan Wusheng,Robay Amal,Kebler Catherine,Kreindler James L.,Guttentag Susan,Hubbard Michael J.,Rubenstein Ronald C.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference68 articles.

1. Processing of mutant cystic fibrosis transmembrane conductance regulator is temperature-sensitive

2. The common variant of cystic fibrosis transmembrane conductance regulator is recognized by hsp70 and degraded in a pre-Golgi nonlysosomal compartment.

3. Perturbation of Hsp90 interaction with nascent CFTR prevents its maturation and accelerates its degradation by the proteasome

4. Sodium 4-phenylbutyrate downregulates Hsc70: implications for intracellular trafficking of ΔF508-CFTR

5. Induction of HSP70 promotes ΔF508 CFTR trafficking

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