Insulin and Isoproterenol Have Opposing Roles in the Maintenance of Cytosol pH and Optimal Fusion of GLUT4 Vesicles with the Plasma Membrane
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference48 articles.
1. Trafficking of glucose transporters in 3T3-L1 cells. Inhibition of trafficking by phenylarsine oxide implicates a slow dissociation of transporters from trafficking proteins
2. Use of bismannose photolabel to elucidate insulin-regulated GLUT4 subcellular trafficking kinetics in rat adipose cells. Evidence that exocytosis is a critical site of hormone action.
3. Temporal Separation of Insulin-stimulated GLUT4/IRAP Vesicle Plasma Membrane Docking and Fusion in 3T3L1 Adipocytes
4. Cell surface accessibility of GLUT4 glucose transporters in insulin-stimulated rat adipose cells. Modulation by isoprenaline and adenosine
5. GLUT4 trafficking in insulin-stimulated rat adipose cells: evidence that heterotrimeric GTP-binding proteins regulate the fusion of docked GLUT4-containing vesicles
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2. Inhibition of ATGL in adipose tissue ameliorates isoproterenol-induced cardiac remodeling by reducing adipose tissue inflammation;American Journal of Physiology-Heart and Circulatory Physiology;2021-01-01
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4. Isoproterenol Induced Insulin Resistance Leading to Diabetic Ketoacidosis in Type 1 Diabetes Mellitus;Case Reports in Endocrinology;2018-12-17
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