A Novel Mechanism for NF-κB-activation via IκB-aggregation: Implications for Hepatic Mallory-Denk-Body Induced Inflammation-Reference-Cited by-同舟云学术

A Novel Mechanism for NF-κB-activation via IκB-aggregation: Implications for Hepatic Mallory-Denk-Body Induced Inflammation

Published:2020-12 Issue:12 Volume:19 Page:1968-1986
ISSN:1535-9476
Container-title:Molecular & Cellular Proteomics
language:en
Short-container-title:Molecular & Cellular Proteomics

Author:

Liu Yi,Trnka Michael J.^ORCID,Guan Shenheng,Kwon Doyoung,Kim Do-Hyung,Chen J.-J.,Greer Peter A.,Burlingame A.L.,Correia Maria Almira

Funder

HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases

HHS | NIH | National Institute of General Medical Sciences

Gouvernement du Canada | Canadian Institutes of Health Research

Dr. Miriam and Sheldon G. Adelson Medical Research Foundation

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Analytical Chemistry

Reference83 articles.

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2. p62 Is a common component of cytoplasmic inclusions in protein aggregation diseases;Zatloukal;Am. J. Pathol,2002

3. The Mallory body: morphological, clinical and experimental studies (Part 1 of a literature survey);Jensen;Hepatology,1994

4. Hepatocellar hyalin (Mallory bodies) in long term griseofulvin-treated mice: a new experimental model for the study of hyalin formation;Denk;Lab. Invest,1975

5. Experimental production of Mallory bodies in mice by diet containing 3,5-diethoxycarbonyl-1,4-dihydrocollidine;Yokoo;Gastroenterology,1982

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