Elk-1, C/EBPα, and Pit-1 Confer an Insulin-responsive Phenotype on Prolactin Promoter Expression in Chinese Hamster Ovary Cells and Define the Factors Required for Insulin-increased Transcription
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference34 articles.
1. Activation of Cell-Specific Expression of Rat Growth Hormone and Prolactin Genes by a Common Transcription Factor
2. A pituitary POU domain protein, Pit-1, activates both growth hormone and prolactin promoters transcriptionally.
3. The insulin and cAMP response elements of the prolactin gene are overlapping sequences.
4. Receptor-like Protein-tyrosine Phosphatase α Specifically Inhibits Insulin-increased Prolactin Gene Expression
5. CCAAT/Enhancer-Binding Protein α Is a Physiological Regulator of Prolactin Gene Expression1
Cited by 17 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Insulin-activated Elk-1 recruits the TIP60/NuA4 complex to increase prolactin gene transcription;Molecular and Cellular Endocrinology;2014-01
2. Identification of functional CCAAT/enhancer-binding protein and Ets protein binding sites in the human chorionic somatomammotropin enhancer sequences;Journal of Molecular Endocrinology;2011-07-07
3. A Pit-1 Threonine 220 Phosphomimic Reduces Binding to Monomeric DNA Sites to Inhibit Ras and Estrogen Stimulation of the Prolactin Gene Promoter;Molecular Endocrinology;2010-01
4. Insulin Acts through FOXO3a to Activate Transcription of Plasminogen Activator Inhibitor Type 1;Molecular Endocrinology;2009-10-01
5. Putative transcription factor binding sites and polymorphisms in the proximal promoter of thePRL-Agene in percomorphs and European sea bass (Dicentrarchus labrax);Marine Ecology;2008-09
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