Prion protein stabilizes amyloid-β (Aβ) oligomers and enhances Aβ neurotoxicity in a Drosophila model of Alzheimer's disease-Reference-Cited by-同舟云学术

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Prion protein stabilizes amyloid-β (Aβ) oligomers and enhances Aβ neurotoxicity in a Drosophila model of Alzheimer's disease

Published:2018-08 Issue:34 Volume:293 Page:13090-13099
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Younan Nadine D.,Chen Ko-Fan,Rose Ruth-Sarah,Crowther Damian C.,Viles John H.

Funder

Wellcome Trust

RCUK | Biotechnology and Biological Sciences Research Council (BBSRC)

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference78 articles.

1. The global prevalence of dementia: a systematic review and metaanalysis;Prince;Alzheimers Dementia,2013

2. Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo;Walsh;Nature,2002

3. Diffusible, nonfibrillar ligands derived from Aβ1–42 are potent central nervous system neurotoxins;Lambert;Proc. Natl. Acad. Sci. U.S.A,1998

4. A specific amyloid-β protein assembly in the brain impairs memory;Lesné;Nature,2006

5. Amyloid β-protein toxicity and the pathogenesis of Alzheimer disease;Yankner;J. Biol. Chem,2009

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