Loss of the tumor suppressor BIN1 enables ATM Ser/Thr kinase activation by the nuclear protein E2F1 and renders cancer cells resistant to cisplatin-Reference-Cited by-同舟云学术

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Loss of the tumor suppressor BIN1 enables ATM Ser/Thr kinase activation by the nuclear protein E2F1 and renders cancer cells resistant to cisplatin

Published:2019-04 Issue:14 Volume:294 Page:5700-5719
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Folk Watson P.,Kumari Alpana,Iwasaki Tetsushi,Pyndiah Slovénie,Johnson Joanna C.,Cassimere Erica K.,Abdulovic-Cui Amy L.,Sakamuro Daitoku

Funder

HHS | NIH | U.S. National Library of Medicine

Augusta University

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference60 articles.

1. Inhibition of cell division in Escherichia coli by electrolysis products from a platinum electrode;Rosenberg;Nature,1965

2. The inhibition of growth or cell division in Escherichia coli by different ionic species of platinum(IV) complexes;Rosenberg;J. Biol. Chem,1967

3. Platinum compounds: a new class of potent antitumour agents;Rosenberg;Nature,1969

4. Cis-diamminedichloroplatinum, vinblastine, and bleomycin combination chemotherapy in disseminated testicular cancer;Einhorn;Ann. Intern. Med,1977

5. The role of cis-platinum in solid-tumor therapy;Einhorn;N. Engl. J. Med,1979

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