Phosphorylation of the HCN channel auxiliary subunit TRIP8b is altered in an animal model of temporal lobe epilepsy and modulates channel function-Reference-Cited by-同舟云学术

Phosphorylation of the HCN channel auxiliary subunit TRIP8b is altered in an animal model of temporal lobe epilepsy and modulates channel function

Published:2019-10 Issue:43 Volume:294 Page:15743-15758
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Foote Kendall M.^ORCID,Lyman Kyle A.,Han Ye,Michailidis Ioannis E.,Heuermann Robert J.,Mandikian Danielle,Trimmer James S.^ORCID,Swanson Geoffrey T.^ORCID,Chetkovich Dane M.

Funder

HHS | National Institutes of Health

Vanderbilt Institute for Clinical and Translational Research

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

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5. Acquired dendritic channelopathy in temporal lobe epilepsy;Bernard;Science,2004

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