Hypertrophic cardiomyopathy mutations increase myofilament Ca2+ buffering, alter intracellular Ca2+ handling, and stimulate Ca2+-dependent signaling-Reference-Cited by-同舟云学术

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Hypertrophic cardiomyopathy mutations increase myofilament Ca2+ buffering, alter intracellular Ca2+ handling, and stimulate Ca2+-dependent signaling

Published:2018-07 Issue:27 Volume:293 Page:10487-10499
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Robinson Paul,Liu Xing,Sparrow Alexander,Patel Suketu,Zhang Yin-Hua,Casadei Barbara,Watkins Hugh,Redwood Charles

Funder

British Heart Foundation (BHF)

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference49 articles.

1. HCM in the general population;Maron;Circulation,1996

2. Sudden death in young athletes;Maron;N. Engl. J. Med,2003

3. Inherited cardiomyopathies;Watkins;N. Engl. J. Med,2011

4. Effects of two hypertrophic cardiomyopathy mutations in α-tropomyosin, Asp175Asn and Glu180Gly, on Ca2+ regulation of thin filament motility;Bing;Biochem. Biophys. Res. Commun,1997

5. Alterations in thin filament regulation induced by a human cardiac troponin T mutant that causes dilated cardiomyopathy are distinct from those induced by troponin T mutants that cause hypertrophic cardiomyopathy;Robinson;J. Biol. Chem,2002

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