Oxidative Modifications and Down-regulation of Ubiquitin Carboxyl-terminal Hydrolase L1 Associated with Idiopathic Parkinson's and Alzheimer's Diseases-Reference-Cited by-同舟云学术

Oxidative Modifications and Down-regulation of Ubiquitin Carboxyl-terminal Hydrolase L1 Associated with Idiopathic Parkinson's and Alzheimer's Diseases

Published:2004-03 Issue:13 Volume:279 Page:13256-13264
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Choi Joungil,Levey Allan I.,Weintraub Susan T.,Rees Howard D.,Gearing Marla,Chin Lih-Shen,Li Lian

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference39 articles.

1. Alzheimer's Disease and Parkinson's Disease

2. Role of ubiquitin-mediated proteolysis in the pathogenesis of neurodegenerative disorders

3. Molecular Pathways of Neurodegeneration in Parkinson's Disease

4. +1 Proteins and aging

5. Oxidatively modified proteins in aging and disease1,2 1Guest Editor: Earl Stadtman 2This article is part of a series of reviews on “Oxidatively Modified Proteins in Aging and Disease.” The full list of papers may be found on the homepage of the journal.

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