Carbon Monoxide Inhibition of Apoptosis during Ischemia-Reperfusion Lung Injury Is Dependent on the p38 Mitogen-activated Protein Kinase Pathway and Involves Caspase 3-Reference-Cited by-同舟云学术

Carbon Monoxide Inhibition of Apoptosis during Ischemia-Reperfusion Lung Injury Is Dependent on the p38 Mitogen-activated Protein Kinase Pathway and Involves Caspase 3

Published:2003-01 Issue:2 Volume:278 Page:1248-1258
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Zhang Xuchen,Shan Peiying,Otterbein Leo E.,Alam Jawed,Flavell Richard A.,Davis Roger J.,Choi Augustine M.K.,Lee Patty J.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference69 articles.

1. Apoptosis induced by ischemia and reperfusion in experimental lung transplantation

2. Functional human heme oxygenase has a neuroprotective effect on adult rat ganglion cells after pressure-induced ischemia

3. Protective effect of heme oxygenase induction in ischemic acute renal failure

4. Upregulation of heme oxygenase-1 protects genetically fat Zucker rat livers from ischemia/reperfusion injury

5. Paradoxical rescue from ischemic lung injury by inhaled carbon monoxide driven by derepression of fibrinolysis

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