Src Kinase Activity Is Essential for Osteoclast Function
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference35 articles.
1. Targeted disruption of the c-src proto-oncogene leads to osteopetrosis in mice
2. Osteoclasts express high levels of pp60c-src in association with intracellular membranes.
3. Osteopetrosis in Src-deficient mice is due to an autonomous defect of osteoclasts.
4. Cbl Associates with Pyk2 and Src to Regulate Src Kinase Activity, αvβ3 Integrin-Mediated Signaling, Cell Adhesion, and Osteoclast Motility
5. Rescue of osteoclast function by transgenic expression of kinase-deficient Src insrc−/− mutant mice
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