Glycosylphosphatidylinositol anchor modification machinery deficiency is responsible for the formation of pro-prion protein (PrP) in BxPC-3 cells and increases cancer cell motility.-Reference-Cited by-同舟云学术

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Glycosylphosphatidylinositol anchor modification machinery deficiency is responsible for the formation of pro-prion protein (PrP) in BxPC-3 cells and increases cancer cell motility.

Published:2016-03 Issue:13 Volume:291 Page:6785
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Yang Liheng,Gao Zhenxing,Hu Lipeng,Wu Guiru,Yang Xiaowen,Zhang Lihua,Zhu Ying,Wong Boon-Seng,Xin Wei,Sy Man-Sun,Li Chaoyang

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Cited by 7 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Protocol to differentiate glycosylphosphatidylinositol-anchored prion protein from pro-prion protein in cancer cells;STAR Protocols;2023-06

2. Pro-prion, as a membrane adaptor protein for E3 ligase c-Cbl, facilitates the ubiquitination of IGF-1R, promoting melanoma metastasis;Cell Reports;2022-12

3. Cellular Prion Protein Role in Cancer Biology: Is It A Potential Therapeutic Target?;Biomedicines;2022-11-07

4. Pro-Prion is a Membrane Adaptor Protein for E3 Ligase C-Cbl to Ubiquitinate IGF-IR Promoting Melanoma Metastasis;SSRN Electronic Journal;2022

5. The Cellular Prion Protein: A Promising Therapeutic Target for Cancer;International Journal of Molecular Sciences;2020-12-02

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