Inhibition or Activation of Apert Syndrome FGFR2 (S252W) Signaling by Specific Glycosaminoglycans-Reference-Cited by-同舟云学术

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Inhibition or Activation of Apert Syndrome FGFR2 (S252W) Signaling by Specific Glycosaminoglycans

Published:2006-03 Issue:11 Volume:281 Page:6924-6930
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

McDowell Lynda M.,Frazier Beth A.,Studelska Daniel R.,Giljum Kari,Chen Jinghua,Liu Jian,Yu Kai,Ornitz David M.,Zhang Lijuan

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference60 articles.

1. Apert syndrome results from localized mutations of FGFR2 and is allelic with Crouzon syndrome

2. De Novo Alu-Element Insertions in FGFR2 Identify a Distinct Pathological Basis for Apert Syndrome

3. Genotype-phenotype analysis in Apert syndrome suggests opposite effects of the two recurrent mutations on syndactyly and outcome of craniofacial surgery

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