Molecular Basis of IaK Protein Regulation by Oxidation or Chelation-Reference-Cited by-同舟云学术

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Molecular Basis of IaK Protein Regulation by Oxidation or Chelation

Published:1995-02 Issue:8 Volume:270 Page:3638-3641
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Busch Andreas E.,Waldegger Siegfried,Herzer Tobias,Raber Gertraud,Gulbins Erich,Takumi Toru,Moriyoshi Koki,Nakanishi Shigetada,Lang Florian

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference20 articles.

1. Cloning of a Membrane Protein That Induces a Slow Voltage-Gated Potassium Current

2. Expression of a minimal K+ channel protein in mammalian cells and immunolocalization in guinea pig heart.

3. MinK Channels: A Minimal Channel Protein with a Maximal Impact

4. The min K channel underlies the cardiac potassium current IKs and mediates species-specific responses to protein kinase C.

5. The Novel Class III Antiarrhythmics NE-10064 and NE-10133 Inhibit Isk Channels Expressed in Xenopus Oocytes and Iks in Guinea Pig Cardiac Myocytes

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1. Redox Regulation of K⁺ Channel: Role of Thioredoxin;Antioxidants & Redox Signaling;2024-08-28

2. Oxidation of K+ Channels in Aging and Neurodegeneration;Aging and disease;2016

3. Direct evaluation of cellular internalization rates using chromogenic disulfides;Molecular BioSystems;2010

4. Involvement of tyrosine kinase in the hyposmotic stimulation of I Ks in guinea-pig ventricular myocytes;Pflügers Archiv - European Journal of Physiology;2007-12-21

5. Oxidative modification of M-type K+ channels as a mechanism of cytoprotective neuronal silencing;The EMBO Journal;2006-10-05

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