Cellular Stresses Differentially Activate c-Jun N-terminal Protein Kinases and Extracellular Signal-regulated Protein Kinases in Cultured Ventricular Myocytes-Reference-Cited by-同舟云学术

Cellular Stresses Differentially Activate c-Jun N-terminal Protein Kinases and Extracellular Signal-regulated Protein Kinases in Cultured Ventricular Myocytes

Published:1995-12 Issue:50 Volume:270 Page:29710-29717
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Bogoyevitch Marie A.,Ketterman Albert J.,Sugden Peter H.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference70 articles.

1. Regulation of cardiac gene expression during myocardial growth and hypertrophy: molecular studies of an adaptive physiologic response

2. Signal transduction pathways of angiotensin II--induced c-fos gene expression in cardiac myocytes in vitro. Roles of phospholipid-derived second messengers.

3. Ternary complex factors: growth factor regulated transcriptional activators

4. The SRF accessory protein Elk-1 contains a growth factor-regulated transcriptional activation domain

5. The c-fos protein interacts with c-JunAP-1 to stimulate transcription of AP-1 responsive genes

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