A MAPK-positive Feedback Mechanism for BLR1 Signaling Propels Retinoic Acid-triggered Differentiation and Cell Cycle Arrest
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference34 articles.
1. RETINOIC ACID INCREASES AMOUNT OF PHOSPHORYLATED RAF; ECTOPIC EXPRESSION OF cFMS REVEALS THAT RETINOIC ACID-INDUCED DIFFERENTIATION IS MORE STRONGLY DEPENDENT ON ERK2 SIGNALING THAN INDUCED G0 ARREST IS
2. Differentiation-specific expression of a novel G protein-coupled receptor from Burkitt's lymphoma
3. Expression of the G-protein--coupled receptor BLR1 defines mature, recirculating B cells and a subset of T-helper memory cells
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