SLAM-associated Protein Deficiency Causes Imbalanced Early Signal Transduction and Blocks Downstream Activation in T Cells from X-linked Lymphoproliferative Disease Patients-Reference-Cited by-同舟云学术

SLAM-associated Protein Deficiency Causes Imbalanced Early Signal Transduction and Blocks Downstream Activation in T Cells from X-linked Lymphoproliferative Disease Patients

Published:2003-08 Issue:32 Volume:278 Page:29593-29599
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Sanzone Silvia,Zeyda Maximilian,Saemann Marcus D.,Soncini Maddalena,Holter Wolfgang,Fritsch Gerhard,Knapp Walter,Candotti Fabio,Stulnig Thomas M.,Parolini Ornella

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference52 articles.

1. X-Linked Lymphoproliferative Disease: A Progressive Immunodeficiency

2. Inactivating mutations in an SH2 domain-encoding gene in X-linked lymphoproliferative syndrome

3. CD150 Association with Either the SH2-Containing Inositol Phosphatase or the SH2-Containing Protein Tyrosine Phosphatase Is Regulated by the Adaptor Protein SH2D1A

4. SH2D1A and slam protein expression in human lymphocytes and derived cell lines

5. Differential methylation pattern of the X-linked lymphoproliferative (XLP) disease gene SH2D1A correlates with the cell lineage-specific transcription

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