Identification of Tyrosine Residues in Vascular Endothelial Growth Factor Receptor-2/FLK-1 Involved in Activation of Phosphatidylinositol 3-Kinase and Cell Proliferation-Reference-Cited by-同舟云学术

Identification of Tyrosine Residues in Vascular Endothelial Growth Factor Receptor-2/FLK-1 Involved in Activation of Phosphatidylinositol 3-Kinase and Cell Proliferation

Published:2001-05 Issue:21 Volume:276 Page:17686-17692
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Dayanir Volkan,Meyer Rosana D.,Lashkari Kameran,Rahimi Nader

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference39 articles.

1. Failure of blood-island formation and vasculogenesis in Flk-1-deficient mice

2. Glioblastoma growth inhibited in vivo by a dominant-negative Flk-1 mutant

3. Inhibition of vascular endothelial growth factor-induced angiogenesis suppresses tumour growth in vivo

4. Increased hemangioblast commitment, not vascular disorganization, is the primary defect in flt-1 knock-out mice

5. Receptor Chimeras Indicate That the Vascular Endothelial Growth Factor Receptor-1 (VEGFR-1) Modulates Mitogenic Activity of VEGFR-2 in Endothelial Cells

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