The Reversion-inducing Cysteine-rich Protein with Kazal Motifs (RECK) Interacts with Membrane Type 1 Matrix Metalloproteinase and CD13/Aminopeptidase N and Modulates Their Endocytic Pathways
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference42 articles.
1. Regulation of matrix metalloproteinase-9 and inhibition of tumor invasion by the membrane-anchored glycoprotein RECK
2. Involvement of the Sp1 Site in ras-Mediated Downregulation of the RECK Metastasis Suppressor Gene
3. The Membrane-Anchored MMP Inhibitor RECK Is a Key Regulator of Extracellular Matrix Integrity and Angiogenesis
4. Decreased RECK Expression Indicating Proteolytic Imbalance in Prostate Cancer is Associated with Higher Tumor Aggressiveness and Risk of Prostate-Specific Antigen Relapse after Radical Prostatectomy
5. Downregulation of RECK by promoter methylation correlates with lymph node metastasis in non-small cell lung cancer
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1. Non‐cell‐autonomous suppression of tumor growth by RECK in immunocompetent mice;Journal of Cellular Physiology;2024-08-05
2. RECK/GPR124‐driven WNT signaling in pancreatic and gastric cancer cells;Cancer Science;2024-06-26
3. Association between the Reduced Expression of RECK and Neutrophilic Inflammation in Chronic Obstructive Pulmonary Disease;International Archives of Allergy and Immunology;2024
4. Pancreatic RECK inactivation promotes cancer formation, epithelial-mesenchymal transition, and metastasis;Journal of Clinical Investigation;2023-09-15
5. Association between the reduced expression of RECK and neutrophilic inflammation in chronic obstructive pulmonary disease;2023-07-21
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