Uncoupling Protein 2, in Vivo Distribution, Induction upon Oxidative Stress, and Evidence for Translational Regulation-Reference-Cited by-同舟云学术

Uncoupling Protein 2, in Vivo Distribution, Induction upon Oxidative Stress, and Evidence for Translational Regulation

Published:2001-03 Issue:12 Volume:276 Page:8705-8712
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Pecqueur Claire,Alves-Guerra Marie-Clotilde,Gelly Chantal,Lévi-Meyrueis Corinne,Couplan Elodie,Collins Sheila,Ricquier Daniel,Bouillaud Frédéric,Miroux Bruno

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference60 articles.

1. Phylogenetic Classification of the Mitochondrial Carrier Family ofSaccharomyces cerevisiae

2. Molecular aspects of isolated and reconstituted carrier proteins from animal mitochondria

3. Cloning and Characterization of an Uncoupling Protein Homolog: A Potential Molecular Mediator of Human Thermogenesis

4. Uncoupling protein-2: a novel gene linked to obesity and hyperinsulinemia

5. Uncoupling Protein-3 Is a Mediator of Thermogenesis Regulated by Thyroid Hormone, β3-Adrenergic Agonists, and Leptin

Cited by 415 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. UCP2, a Member of the Mitochondrial Uncoupling Proteins: An Overview from Physiological to Pathological Roles;Biomedicines;2024-06-13

2. UCP2 polymorphisms, daily step count, and number of teeth associated with all-cause mortality risk in Sado City: A hospital-based cohort study;Heliyon;2024-06

3. Genetic variants in DBC1, SIRT1, UCP2 and ADRB2 as potential biomarkers for severe obesity and metabolic complications;Frontiers in Genetics;2024-05-22

4. Mitochondrial regulation of diabetic endothelial dysfunction: Pathophysiological links;The International Journal of Biochemistry & Cell Biology;2024-05

5. A New Strategy for Targeting UCP2 to Modulate Glycolytic Reprogramming as a Treatment for Sepsis A New Strategy for Targeting UCP2;Inflammation;2024-03-02