Sp1 Is Up-regulated in Cellular and Transgenic Models of Huntington Disease, and Its Reduction Is Neuroprotective-Reference-Cited by-同舟云学术

Sp1 Is Up-regulated in Cellular and Transgenic Models of Huntington Disease, and Its Reduction Is Neuroprotective

Published:2006-06 Issue:24 Volume:281 Page:16672-16680
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Qiu Zhihua,Norflus Fran,Singh Bhupinder,Swindell Mary K.,Buzescu Rodica,Bejarano Michelle,Chopra Raman,Zucker Birgit,Benn Caroline L.,DiRocco Derek P.,Cha Jang-Ho J.,Ferrante Robert J.,Hersch Steven M.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference47 articles.

1. Trinucleotide Repeats in Neurogenetic Disorders

2. Sp1 and TAFII130 Transcriptional Activity Disrupted in Early Huntington's Disease

3. Interference by Huntingtin and Atrophin-1 with CBP-Mediated Transcription Leading to Cellular Toxicity

4. Decreased expression of striatal signaling genes in a mouse model of Huntington's disease

5. Histone deacetylase inhibitors arrest polyglutamine-dependent neurodegeneration in Drosophila

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