A case of acute cardioembolic stroke with Fabry disease treated with intravenous rt-PA
Author:
Affiliation:
1. Department of Neurology, Saitama Medical Center, Saitama Medical University
Publisher
Japan Stroke Society
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/jstroke/40/2/40_10515/_pdf
Reference10 articles.
1. 1) Desnick RJ, Wasserstein MP, Banikazemi M : α-galactosidase A deficiency: Fabry disease. In: Scriver CR, Beaudet AL, Sly WS, Valle S(eds), The metabolic and molecular bases of inherited disease. 8th ed. New York, McGraw-Hill, 2001, pp3733–3774
2. 3) Kawano H, Hirano T, Nakajima M, et al: Modified ASPECTS for DWI including deep white matter lesions predicts subsequent intracranial hemorrhage. J Neurol 259: 2045–2052, 2012
3. 7) DeGraba T, Azhar S, Dignat-George F, et al: Profile of endothelial and leukocyte activation in Fabry patients. Ann Neurol 47: 229–233, 2000
4. 8) Wolf ME, Grittner U, Böttcher T, et al: Phenotypic ASCO Characterisation of Young Patients with Ischemic Stroke in the Prospective Multicentre Observational sifap1 Study. Cerebrovasc Dis 40: 129–135, 2015
5. 13) Mitsias P, Levine SR: Cerebrovascular complications of Fabry’s disease. Ann Neurol 40: 8–17, 1996
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