Bis is Induced by Oxidative StressviaActivation of HSF1-Reference-Cited by-同舟云学术

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Bis is Induced by Oxidative StressviaActivation of HSF1

Published:2014 Issue:5 Volume:18 Page:403
ISSN:1226-4512
Container-title:The Korean Journal of Physiology & Pharmacology
language:en
Short-container-title:Korean J Physiol Pharmacol

Author:

Yoo Hyung Jae¹,Im Chang-Nim¹²³,Youn Dong-Ye¹,Yun Hye Hyeon¹²,Lee Jeong-Hwa¹²³

Affiliation:

1. Department of Biochemistry, College of Medicine, The Catholic University of Korea, Seoul 137-701, Korea.

2. Cancer Research Institute, College of Medicine, The Catholic University of Korea, Seoul 137-701, Korea.

3. Cancer Evolution Research Center, College of Medicine, The Catholic University of Korea, Seoul 137-701, Korea.

Publisher

The Korean Physiological Society and The Korean Society of Pharmacology

Subject

Pharmacology,Physiology

Link

https://synapse.koreamed.org/pdf/10.4196/kjpp.2014.18.5.403

Reference36 articles.

1. Bis, a Bcl-2-binding protein that synergizes with Bcl-2 in preventing cell death

2. An Evolutionarily Conserved Family of Hsp70/Hsc70 Molecular Chaperone Regulators

3. CAIR-1/BAG-3 forms an EGF-regulated ternary complex with phospholipase C-γ and Hsp70/Hsc70

4. CAIR-1/BAG-3 Abrogates Heat Shock Protein-70 Chaperone Complex-mediated Protein Degradation

5. CAIR-1/BAG-3 modulates cell adhesion and migration by downregulating activity of focal adhesion proteins

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1. Hypoxia-Inducible Factor-Dependent and Independent Mechanisms Underlying Chemoresistance of Hypoxic Cancer Cells;Cancers;2024-04-29

2. SRSF3 Is a Critical Requirement for Inclusion of Exon 3 of BIS Pre-mRNA;Cells;2020-10-19

3. Effect of BIS depletion on HSF1-dependent transcriptional activation in A549 non-small cell lung cancer cells;The Korean Journal of Physiology & Pharmacology;2018

4. ‘Molecular habituation’ as a potential mechanism of gradual homeostatic loss with age;Mechanisms of Ageing and Development;2018-01

5. BIS overexpression does not affect the sensitivity of HEK 293T cells against apoptosis;Molecular & Cellular Toxicology;2017-03

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