Mitochondrial dysfunction reduces the activity of KIR2.1 K+ channel in myoblasts via impaired oxidative phosphorylation-Reference-Cited by-同舟云学术

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Mitochondrial dysfunction reduces the activity of KIR2.1 K+ channel in myoblasts via impaired oxidative phosphorylation

Published:2018 Issue:6 Volume:22 Page:697
ISSN:1226-4512
Container-title:The Korean Journal of Physiology & Pharmacology
language:en
Short-container-title:Korean J Physiol Pharmacol

Author:

Woo JooHan¹,Kim Hyun Jong²³,Nam Yu Ran²³,Kim Yung Kyu²,Lee Eun Ju⁴,Choi Inho⁴,Kim Sung Joon¹,Lee Wan³⁵,Nam Joo Hyun²³

Affiliation:

1. Department of Biomedical Sciences, Seoul National University College of Medicine, Seoul 03080, Korea.

2. Department of Physiology, Dongguk University College of Medicine, Gyeongju 38066, Korea.

3. Channelopathy Research Center (CRC), Dongguk University College of Medicine, Goyang 10326, Korea.

4. Department of Medical Biotechnology, Yeungnam University, Gyeongsan 38541, Korea.

5. Department of Biochemistry, Dongguk University College of Medicine, Gyeongju 38066, Korea.

Publisher

The Korean Physiological Society and The Korean Society of Pharmacology

Subject

Pharmacology,Physiology

Link

https://synapse.koreamed.org/pdf/10.4196/kjpp.2018.22.6.697

Reference27 articles.

1. Myoblast fusion: lessons from flies and mice

2. Skeletal muscle formation in vertebrates

3. Fusion of chick embryo skeletal myoblasts: Role of calcium influx preceding membrane union

4. TRANSCRIPTIONAL CONTROL OF MUSCLE DEVELOPMENT BY MYOCYTE ENHANCER FACTOR-2 (MEF2) PROTEINS

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