MicroRNA-10b Plays a Role in Bone Formation by Suppressing Interleukin-22 in Ankylosing Spondylitis
Author:
Affiliation:
1. Department of Rheumatology, Research Institute of Medical Sciences, Chonnam National University Medical School and Hospital, Gwangju, Korea.
2. Department of Rheumatology, Hanyang University Hospital for Rheumatic Diseases, Seoul, Korea.
Publisher
Korean College of Rheumatology
Link
https://synapse.koreamed.org/pdf/10.4078/jrd.2020.27.1.61
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1. A wider spectrum of spondyloarthropathies
2. Interleukin-22, a TH17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis
3. Th17 Cells Expressing KIR3DL2+ and Responsive to HLA-B27 Homodimers Are Increased in Ankylosing Spondylitis
4. Increased numbers of circulating polyfunctional Th17 memory cells in patients with seronegative spondylarthritides
5. Levels of Circulating Th17 Cells and Regulatory T Cells in Ankylosing Spondylitis Patients with an Inadequate Response to Anti−TNF-α Therapy
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