A lead role for a “secondary” axonal injury response-Reference-Cited by-同舟云学术

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A lead role for a “secondary” axonal injury response

Published:2024-05-13 Issue:2 Volume:20 Page:469-470
ISSN:1673-5374
Container-title:Neural Regeneration Research
language:en
Short-container-title:

Author:

Rudy Melissa A.¹²^ORCID,Watkins Trent A.¹^ORCID

Affiliation:

1. Division of Neuroimmunology and Glial Biology, Department of Neurology, University of California at San Francisco, San Francisco, CA, USA (Rudy MA, Watkins TA)

2. Development, Disease Models, and Therapeutics Graduate Program, Baylor College of Medicine, Houston, TX, USA (Rudy MA)

Publisher

Medknow

Reference13 articles.

1. The effect of Jun dimerization on neurite outgrowth and motif binding;Danzi;Mol Cell Neurosci,2018

2. RGC-specific ATF4 and/or CHOP deletion rescues glaucomatous neurodegeneration and visual function;Fang;Mol Ther Nucleic Acids,2023

3. ATF4 leads to glaucoma by promoting protein synthesis and ER client protein load;Kasetti;Nat Commun,2020

4. Activating transcription factor 3 (ATF3) protects retinal ganglion cells and promotes functional preservation after optic nerve crush;Kole;Invest Ophthalmol Vis Sci,2020

5. Dual leucine zipper kinase-dependent PERK activation contributes to neuronal degeneration following insult;Larhammar;Elife,2017

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