Role of peripheral amyloid-β aggregates in Alzheimer’s disease: mechanistic, diagnostic, and therapeutic implications
Author:
Affiliation:
1. Department of Neurology, The University of Texas Health Science Center at Houston, Houston, TX, USA (Gamez N, Morales R)
2. Centro Integrativo de Biologia y Quimica Aplicada (CIBQA), Universidad Bernardo O’Higgins, Santiago, Chile (Morales R)
Publisher
Medknow
Reference18 articles.
1. Iatrogenic Alzheimer’s disease in recipients of cadaveric pituitary-derived growth hormone;Banerjee;Nat Med,2024
2. Peripheral anti-A beta antibody alters CNS and plasma A beta clearance and decreases brain A beta burden in a mouse model of Alzheimer’s disease;DeMattos;Proc Natl Acad Sci U S A,2001
3. The prion-like phenomenon in Alzheimer’s disease: evidence of pathology transmission in humans;Gomez-Gutierrez;PLoS Pathog,2020
4. Peritoneal dialysis reduces amyloid-beta plasma levels in humans and attenuates Alzheimer-associated phenotypes in an APP/PS1 mouse model;Jin;Acta Neuropathol,2017
5. Amyloid β-protein deposition in tissues other than brain in Alzheimer’s disease;Joachim;Nature,1989
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