Biochemical consequences of glucocerebrosidase 1 mutations in Parkinson’s disease-Reference-Cited by-同舟云学术

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Biochemical consequences of glucocerebrosidase 1 mutations in Parkinson’s disease

Published:2023-09-04 Issue:4 Volume:19 Page:725-727
ISSN:1673-5374
Container-title:Neural Regeneration Research
language:en
Short-container-title:

Author:

Yoon Jeong Hyun¹²,Lee Chiao-Yin¹³,Schapira Anthony HV¹³^ORCID

Affiliation:

1. Department of Clinical and Movement Neurosciences, University College London Institute of Neurology, London, UK (Yoon JH, Lee CY, Schapira AHV)

2. Faculty of Medicine, Imperial College London, London, UK (Yoon JH)

3. Aligning Science Across Parkinson’s (ASAP) Collaborative Research Network, Chevy Chase, MD, USA (Lee CY, Schapira AHV)

Publisher

Medknow

Subject

Developmental Neuroscience

Reference15 articles.

1. Chaperone-mediated autophagy markers in Parkinson disease brains;Alvarez-Erviti;Arch Neurol,(2010)

2. Sphingolipid changes in Parkinson L444P GBA mutation fibroblasts promote alpha-synuclein aggregation;Galvagnion;Brain,(2022)

3. Differential effects of severe vs mild GBA mutations on Parkinson disease;Gan-Or;Neurology,(2015)

4. Glucocerebrosidase is present in alpha-synuclein inclusions in Lewy body disorders;Goker-Alpan;Acta Neuropathol,(2010)

5. International Genetics of Parkinson Disease Progression. (IGPP) Consortium (2016) Specifically neuropathic Gaucher's mutations accelerate cognitive decline in Parkinson's;Liu;Ann Neurol

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