MAP4K inhibition as a potential therapy for amyotrophic lateral sclerosis
Author:
Affiliation:
1. Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX, USA
2. Hamon Center for Regenerative Science and Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA
Publisher
Medknow
Subject
Developmental Neuroscience
Reference12 articles.
1. Development of MAP4 kinase inhibitors as motor neuron-protecting agents;Bos;Cell Chem Biol,2019
2. Mitochondrial aging defects emerge in directly reprogrammed human neurons due to their metabolic profile;Kim;Cell Rep,2018
3. The Ste20 family kinases MAP4K4, MINK1, and TNIK converge to regulate stress-induced JNK signaling in neurons;Larhammar;J Neurosci,2017
4. Direct lineage reprogramming reveals disease-specific phenotypes of motor neurons from human ALS patients;Liu;Cell Rep,2016
5. Chemical screens in aging-relevant human motor neurons identify MAP4Ks as therapeutic targets for amyotrophic lateral sclerosis;Liu;bioRxiv,2023
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